Novel strategies targeting residual risk: The promise of PCSK9 inhibiting therapies
10' education - Aug. 28, 2016 - ESC 2016, Rome - Erik Stroes, MD – Academic Medical Center, Amsterdam, The Netherlands - PACE-CME symposium held at ESC 2016 in RomeThis lecture was part of a CME accredited symposium: How to address residual risk post ACS: LDL-c, dyslipidemia, and inflammation held at ESC 2016 in Rome
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Why LDL-C as target to reduce residual risk? 00:14
Why do we need further LDL-C lowering therapies? 02:32
The promise of PCSK9-inhibiting therapies 03:55
Ongoing phase III trials with PCSK9 inhibitors including ACS patients 09:45
The relation between PCSK9 inhibition and inflammation 10:10
Take home messages 13:48
Educational information
The educational objectives of this symposum were to:
- Discuss the potential of current and future strategies targeting inflammation in reducing residual risk in post ACS patients
- Outline current ongoing clinical development programs aimed at testing the inflammation hypothesis of cardiovascular disease
- Understand the unmet need for additional LDL-C-lowering therapies beyond current optimal statin-based therapy as a strategy for addressing lipid-related CV risk in patients post ACS
- Describe the potential impact of PCSK9-based therapies in development in patients who require additional LDL-C reduction
- To review the clinical significance of additional lipid lowering pathways, such as ezetimibe, in the management of patients with ACS
- Understand the implications of new guidelines for lipid management
Disclosures
Prof. Erik Stroes, MD: - Chairman, Department of Vascular Medicine, Academic Medical Center, University of Amsterdam - Amsterdam, The Netherlands
Funding
This EBAC accredited symposium was funded by unrestricted educational grants received from Novartis, MSD, Amgen
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